Pasler F.A., Hassell T.'s Color Atlas of Dental Medicine. Radiology PDF

By Pasler F.A., Hassell T.

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Taraboulos A, Jendroska K, Serban D, Yang SL, DeArmond SJ, Prusiner SB. 1992. Regional mapping 20 PATHWAYS OF CLINICAL FUNCTION AND DISABILITY of prion proteins in brain. Proc Natl Acad Sci. A. 89:7620–7624. Telling GC, Parchi P, DeArmond SJ et al. 1996. Evidence for the conformation of the pathologic isoform of the prion protein enciphering and propagating prion diversity. Science. 274:2079–2082. Tobler I, Gaus SE, Deboer T et al. 1996. Altered circadian activity rhythms and sleep in mice devoid of prion protein.

1997b. Diagnosis of new variant Creutzfeldt–Jakob disease by tonsil biopsy. Lancet. 349:99–100. Hirsch N, Beckett A, Collinge J, Scaravilli F, Tabrizi S, Berry S. 2005. Lymphocyte contamination of laryngoscope blades--a possible vector for transmission of variant Creutzfeldt–Jakob disease. Anaesthesia. 60:664–667. Houston F, Foster JD, Chong A, Hunter N, BostockC J. 2000. Transmission of BSE by blood transfusion in sheep. Lancet. 356:999–1000. Hsiao K, Baker HF, Crow TJ et al. 1989. Linkage of a prion protein missense variant to Gerstmann–Straussler syndrome.

Also, numerous studies have suggested a prominent role of PKC in VSM contraction (Nishizuka 1992; Horowitz, Menice, Laporte et al. 1996; Kanashiro, Khalil 1998; Dallas, Khalil 2003). The most direct evidence is that DAG analogues and phorbol esters, known activators of PKC, cause significant contraction in vascular segments isolated from various blood vessels and examined ex vivo (Khalil, van Breemen 1988; Horowitz, Menice, Laporte et al. 1996; Kanashiro, Khalil 1998). Interestingly, the phorbol ester–induced vascular contraction may not be associated with detectable increases in [Ca2+]i, suggesting the involvement of a Ca2+ -independent PKC isoform such as ε-PKC (Jiang, Morgan 1987; Khalil, Lajoie, Resnick et al.

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Color Atlas of Dental Medicine. Radiology by Pasler F.A., Hassell T.

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